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- Heart muscle cell grafts suppress arrhythmias after heart attacks in animal study
- White blood cells mediate insulin resistance: Neutrophils' role is a surprise and a potential new target for treating diabetes
- Sequencing of malaria genomes reveals challenges, opportunities in battle against parasite
- Link between cell division and growth rate: Puzzling question of how cells know when to progress through the cell cycle answered
- New mechanism behind resistance to cancer treatment that could lead to better therapies
- Aurka-to-p53 signaling: A link between stem cell regulation and cancer
| Heart muscle cell grafts suppress arrhythmias after heart attacks in animal study Posted: 05 Aug 2012 11:48 AM PDT Researchers have made a major advance in efforts to regenerate damaged hearts. Grafts of human heart muscle cells reduced the incidence of irregular heart rhythms after heart attacks in animal studies. Grown from embryonic stem cells, the grafted cells couple electrically and contract in sync with the heart's own muscle. The results offer evidence that human heart muscle cell grafts meet physiological criteria for true heart regeneration. |
| Posted: 05 Aug 2012 11:48 AM PDT Researchers say neutrophils, an abundant type of white blood cell typically tasked with attacking bacteria and other foreign invaders, also plays an unexpected role in mediating insulin resistance -- the central characteristic of type 2 diabetes, which afflicts an estimated 26 million Americans. |
| Sequencing of malaria genomes reveals challenges, opportunities in battle against parasite Posted: 05 Aug 2012 11:48 AM PDT Genetic variability revealed in malaria genomes newly sequenced by two multi-national research teams points to new challenges in efforts to eradicate the parasite, but also offers a better picture of its genetic composition, providing an initial roadmap in the development of vaccines to combat malaria. |
| Posted: 05 Aug 2012 11:48 AM PDT It's a longstanding question in biology: How do cells know when to progress through the cell cycle? Researchers have now precisely measured the growth rates of single cells, allowing them to answer that fundamental question. They report that mammalian cells divide not when they reach a critical size, but when their growth rate hits a specific threshold. |
| New mechanism behind resistance to cancer treatment that could lead to better therapies Posted: 05 Aug 2012 11:48 AM PDT Developing resistance to chemotherapy is a nearly universal, ultimately lethal consequence for cancer patients with solid tumors – such as those of the breast, prostate, lung and colon – that have metastasized, or spread, throughout the body. A team of scientists has discovered a key factor that drives this drug resistance – information that ultimately may be used to improve the effectiveness of therapy and buy precious time for patients with advanced cancer. |
| Aurka-to-p53 signaling: A link between stem cell regulation and cancer Posted: 03 Aug 2012 08:08 AM PDT Researchers have found a new role for an oncogenic signaling pathway in embryonic stem cell (ESC) self-renewal and in reprogramming adult cells into an ESC-state, which will aid in the development of future cancer therapies. |
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